소뇌경색 : 27례에서 임상소견과 방사선소견의 연관성 |
조상걸, 오건세,정항재,안무영,신현길,이광호,김대호 |
순천향대학교 신경과, 진단방사선과. |
Creabellar Infarction : A Clinicoradiologic Correlation of 27 Cases |
Sang-Gull Cho, M.D., Gun-Sei Oh, M.D., Jang-Je Chung, M.D., Mu-Young Ahn, M.D., Hyun-Gil Shin, M.D., Kwang-Ho Lee, M.D., Dae-Ho Kim, M.D. |
Department of Neurology and Radiology, College of Medicine Soonchunghyang University |
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Abstract |
We reviewed 27 patients wlth cerebellar infarction which was demonstrated by brain CT and/or MRI. Infarction occurred in the territory of posterior inferior cerebellar artery (PICAj in 16 patients, and the territory of the superior cerebellar artery(SCA) was involved in 5 patients. Antenor inferior cerebellar artery(AICA) infarcts occurred in 3 patients. Both PICA and SCA temtories were involved in 2 patients. In the remaining 1 patient, the infarct encompassed the borderzone between the SCA and PICA territories. The main symptoms and signs were sudden onset of vertigo, dizziness, nausea, vomiting, dysmetria, ataxia, nystagmus, and headache. There were signs of associated brain stem infarction or occipitotemporal infarction ;rostral basilar artery syndrome, classic SCA syndrome, Wallenberg syndrome, internuclear ophthalmoplegia, facial palsy, hearing impairment. Presumed cerebral embolism was the main stroke mechanism in the SCA terntories. Six patients with brainstem compression or brainstem involvement showed consciousness deterioration, and only one of them died as a result of extensive cerebellar infarctions involving both SCA and PICA territories Cerebellar infarction may run a more benign course than previously thought . |
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